Horseshoe kidney structural abnormalities of the kidney disease , the majority of patients with horseshoe kidney clear understanding that some life to the effect that it also makes horseshoe kidney disease has been increasing , leading to some complications. Therefore, understanding horseshoe kidney , horseshoe kidney understand the majority of the relevant considerations in patients with horseshoe kidney has a very important role.
Etiology and pathology
Both sides of the upper pole of the kidney or lower pole blending synthetic horseshoe kidney . Horseshoe kidney occurs in the early embryo , the embryo is in the umbilical artery on both sides of the kidney is tightly squeezed between the fusion results.
Clinical
Most patients due to nerve plexus, blood circulation or ureter oppressed and hair symptoms. There are upper abdomen, umbilical or lower back pain, chronic constipation, and urinary symptoms , such as chronic nephritis, pyelonephritis , hydronephrosis and calculi.
Imaging Findings
1 , KUB and angiography: According to bilateral renal shadow near the spine and lows , the long axis parallel to the upper pole or lower pole tilted outwards inwards close to suspect horseshoe kidney , sometimes visible connecting the lower pole renal isthmus contours. Urography can confirm the diagnosis , the most obvious signs is the next destination point calyx centerline axis upper calyceal renal pelvis inward outward bottom inverted " eight " shape, ureter closer to the midline .
2, B Chao: clearly shows two lower pole renal connected across the inferior vena cava and the aorta ahead.
3, CT: can directly display the two lower pole renal fusion unit that crossed the aortic isthmus in front , and because the kidney malrotation , renal calyces located in front of the ureter crossed the isthmus on both sides of the front down. However, the general location of horseshoe kidney lower lower position can be scanned to confirm the diagnosis .
4, MR and radionuclide is also available from most shows two coronal lower pole renal fusion .
Horseshoe kidney complications
1 , horseshoe kidney may be due to repeated infections and poor urine flow and kidney stones .
2 , horseshoe kidney can cause urinary obstruction , urinary obstruction can cause hydronephrosis, uremia bilateral obstruction may occur .
Horseshoe kidney treatment
Because horseshoe kidney is a congenital kidney abnormalities , so horseshoe kidney treatment should be symptomatic treatment . It is important to use traditional Chinese medicine to increase the amount of blood flow , increasing the body's micro- circulation and prevent urinary obstruction . As the traditional Chinese medicine oral irritation of the gastrointestinal tract , is not conducive to traditional Chinese medicine absorption , penetration of Micro-Chinese Medicine recommended treatment .
kidney WE care
kidney self care is very important for us. this blogger is to help us keep a health kidney. hope everyone can find something avaible.
Tuesday, September 24, 2013
Thursday, September 5, 2013
what diet should people with pupura nephritis pay attention to
For people with allergic purpura nephritis,a scientific diet can not only delay the progression of nephritis, and for recovery from disease certainly helps. On one hand it is to prevent further allergies, on the other hand it can protect the kidneys and reduce kidney damage. Specific principles are as follows:
Purpura nephritis patients should first stop then or use may cause food allergies, such as milk, fish, shrimp, crab, mutton.
The different choice of foods should be based on the type of people's physical condition. people with cold physique should use in migraine cold food, such as lotus root, water chestnuts, shepherd's purse, pear, lotus seeds, fungus, lotus leaf , thistle, etc.
If the deficiency is not photographed, should use spleen and kidney, nourishing tonic products, such as meat, skim milk powder, red dates, lotus seeds, etc.
In the clinical treatment of autoimmune kidney disease on (lupus nephritis, nephritis, IGA nephropathy) have a significant effect. The therapy causes kidney disease from the start, quickly clear away cause hypersensitivity immune complexes, repair damaged kidney cells, to correct disorders of the immune system environment in which immune cells dynamic equilibrium state.
Wednesday, September 4, 2013
can IgA nephropathy be cured
Iga nephropathy patients can be cured? We want to treat this disease in many aspects to consider, not with a terms. To understand the etiology of disease, disease stage of development, and then do further identified, in order to determine whether it can be cured.
IgA nephropathy is a mucosal immune deficiency diseases, mainly in the upper respiratory tract infections, gastrointestinal infections, urinary tract infections occurring after, had gross hematuria or more persistent microscopic hematuria, glomerular damage with or without urinary protein. iga iga nephropathy is composed mainly of immune complex deposition resulting from abnormal intrinsic renal cell injury. Renal ischemia and hypoxia, intrinsic renal cell damage and destruction of red blood cells can not be blocked, proteinuria leakage. Early iga nephropathy no standardized restorative treatment, the treatment is relatively good rate is also small, gradual loss of kidney function, the disease will soon progressed to renal insufficiency, uremia. iga nephropathy fundamental - for renal intrinsic cells reversed restorative treatment, while improving the state of renal ischemia and hypoxia.
IgA nephropathy, after years of unremitting efforts and explore, Xi'an Tongji Hospital experts to develop the natural immune balance therapy iga nephropathy, but will help to improve heart function, correction of anemia and blood pressure control, integration with existing kidney disease treatment techniques, forming a iga nephropathy treatment of a stereo system, about 10% -30% of patients with acute nephritis, balanced natural immune system therapy protect and restore renal function. Promote intrinsic renal cell repair and regeneration, early nephropathy reparative therapy is the best way, you can add some trace elements rich in vegetables, grains, fruits, seafood, etc. to be added, more extensive treatment of iga nephropathy prospects.
could diabetic nephropathy be cured
Diabetic nephropathy (DN) is a common clinical and multiple complications of diabetes . In other words , diabetic nephropathy is one of the most serious complications of diabetes , but also the main reason for ESRD . Therefore , patients with diabetes to be highly vigilant diabetic nephropathy, from the outset, to enhance self-care and self-protection , especially from the diet to proceed, reduce kidney stress. If you have already lead to diabetic nephropathy , we must take effective treatment measures , and that diabetic nephropathy in the end can be cured?
Diabetic nephropathy can be cured? Accept the system as long as the early diabetic nephropathy treatment, and follow doctor's orders , and adhere to a reasonable diet to restore objects with the treatment , it can get a good efficacy .
Face treatment of diabetic nephropathy , Xi'an Tongji Hospital kidney specialist team , after 20 years of scientific research , and constantly improve and enhance the "natural immune balance therapy system" outstanding contributions to the treatment of diabetic nephropathy , the formation of a scientific , advanced and sophisticated treatment system.
Diabetic nephropathy can be cured? "Natural immune balance therapy system " for the treatment of diabetic nephropathy , can achieve the desired effect of treating the symptoms , the clinical treatment has four effects:
First, the traditional classical side healer : Shenfukang , kidney Tecom , kidney BaoKang , etc. healer Shenshuaikang capsule treatment in clinical practice to get a good effect. Shenfukang series is based on the classic recipe , based on the theory of immune balance , breaking the traditional material with conventional treatment , screening with a strong main biologically active animals and dozens of kinds of valuable therapeutic substance in the treatment of materials , clever compatibility, carefully group side , the use of modern biotechnology to extract refined series of new therapeutic effects matter , according to the pathological features of diabetic nephropathy in the control of blood sugar while protecting renal function.
Second, the compound liquid : Balancing environment for the body to correct immune disorders , blood circulation, regulate the flow of blood to remove immune complexes and toxins, to clarify the source copy effect. Combined with physical therapy in the treatment of substance West , according to the case in particular for regular treatments and research programs developed and achieved good clinical efficacy.
Third, the ion superconducting partial activation treatment and intracapsular injection : direct lesions , intensive treatment , avoid physical side effects of hormone therapy and renal systemic treatment area barter minimal role of the state.
Fourth, the healer bath fumigation treatments : topical treatment for the prevention of diabetes complications substance bath has a positive effect .
Five , colon to treat things - intestinal dialysis therapy: for patients with impaired renal function , through the intestinal detoxification, has a unique effect .
In addition to the treatment of diabetic nephropathy , the Tongji Hospital of Xi'an kidney specialists recommend that patients should do the following four points:
First, the active and effective control of blood glucose : can play a preventive role in progression of diabetic nephropathy .
Second, the blood pressure lowering treatment : patients with diabetes have high blood pressure and kidney disease benefits , while urea nitrogen and serum creatinine should be monitored , as appropriate, with the treatment thereof.
Third, low-protein diet: moderately low protein diet of diabetic nephropathy , both non- clinical or clinical stage of the patient, can make the urine protein decreased.
Four other Note: In the course of treatment of diabetic nephropathy , to prevent hypoglycemia ; prevent calcium , phosphorus , potassium, sodium metabolism ; restrict the use of contrast agents , the treatment of renal toxicity barter and salt intake .
Sunday, September 1, 2013
what hyperuricemia nephroapthy will bring you
Long-term consequences of hyperuricemia mainly gouty arthritis and kidney damage. Kidney damage after joint disease. Joints often obvious symptoms, and kidney disease is insidious , gout repeated more than 10 years the performance side has kidney damage . Acute hyperuricemia nephropathy mainly acute renal failure .
1 , chronic hyperuricemia nephropathy was acute gouty arthritis , aggravated at night . Often due to mental stress, fatigue, feast, alcoholism and infection -induced . Minor trauma such as surgery can also be induced . Metatarsophalangeal joints in the affected joint as much, followed by the ankle , hand, wrist , knee , etc. Especially in the first metatarsophalangeal joint is most common. Joint pain began to appear after a few hours hypersensitivity and significant redness, swelling, heat, pain . Shoulder, hip and other large joints less involved , but once involved often exudate . Progress with the disease , the deposition of urate in the joints gradually increased after the onset of frequent joint hypertrophy , fibrosis , with joint deformity, stiffness resulting in limited mobility .
Hyperuricemia change nephrotic urine mainly mild proteinuria and a small amount of red blood cells in urine . The early changes in renal function is concentrated function decline , and then gradually affecting glomerular filtration rate . When there is high blood pressure , atherosclerosis , diabetes, renal cysts and amyloid lesions exist, renal failure may occur , but also the fate of gouty nephropathy . Foreign reports gout and hypertension was 40%, the domestic is 63,1 % . Because 25% to 35 % of primary hypertension associated with hyperuricemia, so Hyperuricemia is a risk factor for cardiovascular disease . Hyperuricemia is often accompanied by high blood pressure , obesity and other risk factors of atherosclerosis prone to cause coronary heart disease, may also be urate crystal deposition in the arterial wall and intimal injury related .
2 , acute renal hyperuricemia
Leukemia, lymphoma and other myeloproliferative disorders and malignant tumors are widely disseminated , especially when treated with radiotherapy and chemotherapy, when a large number of hyperuricemia to renal excretion of uric acid , the uric acid crystals in the renal tubules , collecting ducts and renal pelvis abrupt deposition , causing tubular intraluminal pressure increased glomerular capsule pressure increased , resulting in a sharp decline in glomerular filtration rate . The clinical features of early excretion of uric acid increased , how -shaped crystals in urine , hematuria , and a small amount of proteinuria. Lesions progress oliguria and anuria, may be associated with low back pain , nausea , vomiting and lethargy uremia . Hyperuricemia acute uric acid nephropathy and uric acid were significantly increased . Reports in the literature of serum uric acid > 1189μmol / L (20mg/dl) accounted for 60% ; while only 14% of primary gout ; 20% of patients 24h urinary uric acid excretion > 19,48 μmol / L, while only 6 primary gout % .
3 , urate stones 10% to 25% of gout patients have kidney stones , often 200 times higher than the normal population . 1000mg daily uric acid excretion by approximately 20% with stone disease, such as when the discharge 1100mg half stones . Similarly urate stones and hyperuricemia , uric acid 1,37 μmol / L, then 50% of kidney stones. There are a number of diseases not associated with hyperuricemia and excess uric acid excretion , but because of less urine , urine can occur with high uric acid stones . As for ileostomy diarrhea , prostate disease in elderly patients , because of difficulties in urinating and drinking reduced , causing less urine , pH is low, uric acid stones may also occur.
Urate stones in the urinary tract symptoms are local irritation , urinary obstruction and secondary infection. These symptoms are due to stone size , shape , location and presence or absence of infection varies . Urate stones were round or oval , smooth or slightly rough, yellowish brown , solid texture . Renal colic tips obstruction. Few patients have bilateral renal colic ( ie kidney - kidney reflex ) , hematuria after the event , the row of stone . Manifestations of dysuria, urinary flow interrupt and even sudden anuria ( calculus anuria ) . Small stones are available from the urine. Approximately 80% of patients with gout not urate stones . Pure uric acid stones are translucent , usually can not be developed. But more than 2cm in diameter may be calcium oxalate and calcium phosphate stones and other mixed stones , and can be developed. Upper urinary tract calculi make huge renal pelvis deformation.
1 , chronic hyperuricemia nephropathy was acute gouty arthritis , aggravated at night . Often due to mental stress, fatigue, feast, alcoholism and infection -induced . Minor trauma such as surgery can also be induced . Metatarsophalangeal joints in the affected joint as much, followed by the ankle , hand, wrist , knee , etc. Especially in the first metatarsophalangeal joint is most common. Joint pain began to appear after a few hours hypersensitivity and significant redness, swelling, heat, pain . Shoulder, hip and other large joints less involved , but once involved often exudate . Progress with the disease , the deposition of urate in the joints gradually increased after the onset of frequent joint hypertrophy , fibrosis , with joint deformity, stiffness resulting in limited mobility .
Hyperuricemia change nephrotic urine mainly mild proteinuria and a small amount of red blood cells in urine . The early changes in renal function is concentrated function decline , and then gradually affecting glomerular filtration rate . When there is high blood pressure , atherosclerosis , diabetes, renal cysts and amyloid lesions exist, renal failure may occur , but also the fate of gouty nephropathy . Foreign reports gout and hypertension was 40%, the domestic is 63,1 % . Because 25% to 35 % of primary hypertension associated with hyperuricemia, so Hyperuricemia is a risk factor for cardiovascular disease . Hyperuricemia is often accompanied by high blood pressure , obesity and other risk factors of atherosclerosis prone to cause coronary heart disease, may also be urate crystal deposition in the arterial wall and intimal injury related .
2 , acute renal hyperuricemia
Leukemia, lymphoma and other myeloproliferative disorders and malignant tumors are widely disseminated , especially when treated with radiotherapy and chemotherapy, when a large number of hyperuricemia to renal excretion of uric acid , the uric acid crystals in the renal tubules , collecting ducts and renal pelvis abrupt deposition , causing tubular intraluminal pressure increased glomerular capsule pressure increased , resulting in a sharp decline in glomerular filtration rate . The clinical features of early excretion of uric acid increased , how -shaped crystals in urine , hematuria , and a small amount of proteinuria. Lesions progress oliguria and anuria, may be associated with low back pain , nausea , vomiting and lethargy uremia . Hyperuricemia acute uric acid nephropathy and uric acid were significantly increased . Reports in the literature of serum uric acid > 1189μmol / L (20mg/dl) accounted for 60% ; while only 14% of primary gout ; 20% of patients 24h urinary uric acid excretion > 19,48 μmol / L, while only 6 primary gout % .
3 , urate stones 10% to 25% of gout patients have kidney stones , often 200 times higher than the normal population . 1000mg daily uric acid excretion by approximately 20% with stone disease, such as when the discharge 1100mg half stones . Similarly urate stones and hyperuricemia , uric acid 1,37 μmol / L, then 50% of kidney stones. There are a number of diseases not associated with hyperuricemia and excess uric acid excretion , but because of less urine , urine can occur with high uric acid stones . As for ileostomy diarrhea , prostate disease in elderly patients , because of difficulties in urinating and drinking reduced , causing less urine , pH is low, uric acid stones may also occur.
Urate stones in the urinary tract symptoms are local irritation , urinary obstruction and secondary infection. These symptoms are due to stone size , shape , location and presence or absence of infection varies . Urate stones were round or oval , smooth or slightly rough, yellowish brown , solid texture . Renal colic tips obstruction. Few patients have bilateral renal colic ( ie kidney - kidney reflex ) , hematuria after the event , the row of stone . Manifestations of dysuria, urinary flow interrupt and even sudden anuria ( calculus anuria ) . Small stones are available from the urine. Approximately 80% of patients with gout not urate stones . Pure uric acid stones are translucent , usually can not be developed. But more than 2cm in diameter may be calcium oxalate and calcium phosphate stones and other mixed stones , and can be developed. Upper urinary tract calculi make huge renal pelvis deformation.
Thursday, August 29, 2013
Why you have Urate stones
10% ~ 25% gout patients have kidney stone, which is about 200 times higher than normal people. Uric acid eduction 1000 mg a day about 20% have stone, such as discharge 1100 mg nearly half of the stones. Urate stones and high uric acid hematic disease, blood uric acid 1, 37 mu mol/L, 50% had kidney stones. There are some diseases are associated with high blood uric acid and urine uric acid eduction too much, but because of the less amount of urine, urine acid high uric acid calculi can still occur. Such as make the ileum colostomy diarrhea, prostate disease in elderly patients, because of dysuria and less water, less urine output, low pH, uric acid calculi can also occur.
Urate stones mainly have the symptoms of urinary tract local irritation, urine flow obstruction and secondary infection. These symptoms because of the size, shape, site of the stones and presence of infection. Urate stones more assumes the circular or oval, surface smooth or slightly rough, yellowish-brown, quality of a material is solid. With renal colic suggests obstruction. A few patients with bilateral renal colic (i.e., kidney - kidney reflex), hematuria, platoon stone after activity. Performance with dysuria, urinary flow interruption and sudden anuresis (stone in urinary closed). Small stones from the eduction in urine. About 80% patients with gout calculi not uric acid salt. Pure uric acid calculi is pervious to light, usually can't development. But more than 2 cm diameter for calcium oxalate and calcium phosphate mixed stones, generally can be developed. Upper tract huge can make the pelvic calyces deformation.
Saturday, August 24, 2013
relationship between hypertension and diabetes
Diabetes and hypertension are inextricably linked, they are each other "curse", reinforce each other, which is mainly manifested in the following aspects:
① diabetes incidence of hypertension, diabetes, hypertension incidence is much higher than normal blood pressure. Many people are first found high blood pressure, diabetes, and then found that some of diabetic patients found to have high blood pressure at the same time, the first few patients have diabetes, the recurrence of high blood pressure. As in the detection of diabetes gradually increased blood pressure after a few years, you should be careful to check kidney function.
② high blood sugar more insulin to stimulate insulin secretion, resulting in hyperinsulinemia. Excessive insulin not only promotes renal tubular sodium reabsorption, causing sodium retention, but also stimulate the sympathetic nervous excitement, and thus make vasoconstriction, increased peripheral resistance, elevated blood pressure naturally.
③ occurrence and development of diabetic nephropathy can cause and aggravate high blood pressure in people with diabetes, and to target organs such as the heart, blood vessels, eyes, nervous system diseases increased.
④ Some antihypertensive drugs can affect diabetes control, as well as aggravate high blood sugar, such as loss of potassium-sparing diuretics, β-blockers.
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